Pass the ARDMS RDCS AE-Adult-Echocardiography Questions and answers with CertsForce

Intravenous drug use is a major risk factor for infective endocarditis, particularly involving the tricuspid valve and sometimes left-sided valves. Symptoms like fever, flu-like illness, dyspnea, and chest pain suggest possible septic emboli or valve destruction.

Echocardiographic findings associated with endocarditis include mobile echogenic masses attached to valve leaflets (vegetations), valve thickening, or destruction. These findings are diagnostic and guide treatment.

Aortic dissection, hypertrophic cardiomyopathy, and mitral valve prolapse can present with different clinical features and echocardiographic findings not consistent with infectious vegetations.

These clinical and echocardiographic correlations are detailed in the ASE guidelines on infective endocarditis and the "Textbook of Clinical Echocardiography"【16:Textbook of Clinical Echocardiography, 6e†p.470-475】【12:ASE Infective Endocarditis Guidelines†p.380-390】.

Comprehensive and Detailed Explanation From Exact Extract:

Anteroseptal hypokinesis is most often due to ischemia or infarction in the left anterior descending (LAD) artery territory, a major branch of the left coronary artery. The LAD supplies the anterior wall and the interventricular septum.

The right coronary artery generally supplies the inferior wall and right ventricle. The circumflex artery supplies the lateral wall. The posterior descending artery supplies the inferior wall.

This coronary artery distribution and wall motion correlation is fundamental in stress echocardiography and ischemic heart disease assessment as detailed in ASE guidelines and clinical echocardiography references【12:ASE Stress Echocardiography Guidelines†p.300-310】【16:Textbook of Clinical Echocardiography, 6e†p.380-385】.

During dobutamine stress testing, a normal physiological response includes increased myocardial contractility leading to a decrease in left ventricular (LV) cavity size during systole due to more effective ejection. Concurrently, systolic blood pressure increases due to the inotropic and chronotropic effects of dobutamine.

An increase in LV cavity size during stress would suggest impaired contractility or ischemia, which is abnormal.

This normal response is detailed in the "Textbook of Clinical Echocardiography, 6e", Chapter on Stress Echocardiography and Hemodynamic Responses【20:400-405†Textbook of Clinical Echocardiography】.

The video shows a transthoracic echocardiographic apical four-chamber or modified view focusing on the left atrium and adjacent structures. The arrow points to a vessel entering the left atrium from the right side of the image, which corresponds anatomically to the right upper pulmonary vein. The right upper pulmonary vein returns oxygenated blood from the right lung to the left atrium and is visualized in echocardiography as entering the superior-lateral aspect of the left atrium.

The left upper pulmonary vein enters the left atrium on the opposite side. The right and left pulmonary arteries are located anteriorly and superiorly in the mediastinum and are visualized mainly in the parasternal or suprasternal views, not the apical four-chamber.

This identification aligns with standard adult echocardiography anatomy as described in the "Textbook of Clinical Echocardiography" and ASE guidelines on pulmonary vein imaging【12:ASE Pulmonary Vein Imaging Guidelines†p.110-115】【16:Textbook of Clinical Echocardiography, 6e†p.120-125】.

The subcostal echocardiographic view is best obtained with the patient supine, knees bent to relax abdominal muscles, and the patient holding a deep breath at the end of inhalation to lower the diaphragm and improve acoustic window through the subxiphoid area.

Left lateral decubitus position is used for parasternal and apical views but is not optimal for subcostal imaging.

This patient positioning and respiration technique are described in the "Textbook of Clinical Echocardiography, 6e", Chapter on Echocardiographic Windows and Imaging Techniques【20:90-95†Textbook of Clinical Echocardiography】.

Comprehensive and Detailed Explanation From Exact Extract:

Pulmonary capillary wedge pressure (PCWP) is obtained by advancing a balloon-tipped catheter into a small branch of the pulmonary artery and inflating the balloon to "wedge" the catheter, thereby occluding forward blood flow and measuring the pressure distal to the occlusion. The measured pressure reflects the pressure in the pulmonary venous system, which closely approximates left atrial pressure (LAP) under normal conditions.

Since the left atrium receives pulmonary venous return before the blood enters the left ventricle, PCWP is a surrogate for LAP, which in turn reflects left ventricular end-diastolic pressure (LVEDP) in the absence of mitral valve disease or pulmonary venous obstruction. PCWP is widely used in clinical and echocardiographic contexts to estimate left heart filling pressures.

It does not estimate right atrial, right ventricular, or left ventricular pressures directly. Right atrial pressure is measured via central venous pressure, right ventricular pressure by catheterization, and left ventricular pressure by direct catheterization.

This concept is extensively discussed in the "Textbook of Clinical Echocardiography, 6e", Chapter on Hemodynamics and Doppler Assessment, with specific emphasis on the use of PCWP to estimate left atrial pressure【20:200-210†Textbook of Clinical Echocardiography】.

Acute severe aortic regurgitation causes rapid volume overload of the left ventricle during diastole, leading to a marked increase in left ventricular end-diastolic pressure (LVEDP). This elevated LVEDP results from the sudden return of blood into the ventricle and impaired compliance.

End-systolic pressure is not primarily affected. Early-diastolic pressure changes relate to aortic pressure but LVEDP is critical. Early-systolic pressure is not typically affected by AR.

This hemodynamic effect is discussed in the "Textbook of Clinical Echocardiography, 6e", Chapter on Aortic Regurgitation and Hemodynamics【20:375-380†Textbook of Clinical Echocardiography】.

The M-mode image shows characteristic diastolic doming or “hockey stick” appearance of the anterior mitral leaflet with restricted leaflet motion. This is a classic sign of mitral stenosis, where leaflet thickening and fusion cause limited opening during diastole.

Dilated cardiomyopathy shows increased chamber sizes and decreased systolic function but not mitral leaflet doming. Hypertrophic cardiomyopathy is characterized by septal thickening and SAM of the mitral valve. Mitral valve prolapse shows leaflet billowing into the left atrium during systole.

This pattern is well described in ASE valvular heart disease guidelines and echocardiography texts【12:ASE Valve Imaging Guidelines†p.180-185】【16:Textbook of Clinical Echocardiography, 6e†p.200-205】.

The arrow points to the left anterior descending (LAD) coronary artery, which runs in the anterior interventricular groove toward the apex of the heart. It supplies the anterior wall of the left ventricle.

The right coronary artery runs in the right atrioventricular groove. The left main coronary artery is proximal to the LAD and circumflex arteries. The circumflex artery runs in the left atrioventricular groove posteriorly.

This identification is detailed in the "Textbook of Clinical Echocardiography, 6e", Chapter on Coronary Artery Anatomy and Echocardiographic Visualization【20:150-155†Textbook of Clinical Echocardiography】.

Cardiac contusion is a myocardial injury resulting from blunt chest trauma, typically affecting the right ventricle more commonly than the left ventricle because of its anterior location and proximity to the chest wall. The injury can range from mild bruising to severe myocardial damage and dysfunction.

It does not result from myocardial infarction (which is ischemic injury), nor does it cause hypertrophy or hypercontractility. Instead, it may cause wall motion abnormalities, arrhythmias, or even rupture.

These features are detailed in echocardiography and trauma cardiology literature, including the "Textbook of Clinical Echocardiography" and clinical guidelines on blunt cardiac injury【16:Textbook of Clinical Echocardiography, 6e†p.600-605】【12:ASE Trauma Cardiology Guidelines†p.500-505】.